Hawthorn
Crataegus monogyna
Crataegus laevigata
Botanical Classification
Family: Rosaceae (Rose family)
Other Names
Maythorn, Whitethorn, Haw, Thornapple
Fruits: Hawthorn berries
Description
A deciduous shrub growing between 2 and 10 meters tall, with smooth, light gray bark. The branches bear sharp thorns and leathery, lobed leaves. The upper surface of the leaves is glossy dark green, while the underside is bluish to light green.
The mostly white (occasionally pink) flowers have a pleasant amine-like scent that attracts pollinating insects. The flowers grow in corymb clusters at the tips of the branches.
In autumn, round red fruits develop that resemble small rose hips. The fruits are yellow inside and have a mealy texture and taste.
Flowering Time
May to June
Habitat
Native throughout Europe.
Cultivation
Prefers dry soils at forest edges and in light scrubland.
Plant Parts Used
Leaves and flowers.
Taste
Slightly sweet to mildly bitter, mildly astringent.
Traditional Folk Use
In late antiquity, hawthorn was described as helpful for diarrhea and snake bites. During the Middle Ages, it largely fell into obscurity. Later, it was used to promote circulation in the extremities.
Active Compounds
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Dimeric and oligomeric procyanidins (OPC), standardized extracts contain approx. 18.75%
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Flavonoids (hyperoside, rutin, vitexin rhamnoside, eriodictyol glycoside)
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Triterpenic acids (ursolic acid, oleanolic acid, 2‑α‑hydroxy‑oleanolic acid)
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Chlorogenic acid
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Caffeic acid
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Aromatic amines (phenylethylamine, tyramine, 6‑methoxyphenylethylamine, aminopurines, catechins)
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Potassium salts
Mechanism of Action
Effects of Oligomeric Procyanidins and Flavonoids
Hawthorn extracts:
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Increase myocardial contractility (positive inotropic effect)
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Improve electrical conduction (positive dromotropic effect)
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Reduce excitability (negative bathmotropic effect)
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Lower peripheral vascular resistance (afterload reduction)
These effects result in:
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Increased cardiac output
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Improved coronary and myocardial blood flow
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Enhanced tolerance of the heart muscle to oxygen deficiency
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More efficient heart rhythm and performance
Cellular and Molecular Mechanisms
The positive inotropic effect is attributed at the cellular level to:
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Increased calcium influx
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Enhanced intracellular calcium release
Vasodilation is likely related to modulation of potassium-dependent calcium ion channels.
At the molecular level, effects are thought to involve:
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Inhibition of cAMP phosphodiesterase
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Inhibition of angiotensin-converting enzyme (ACE)
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Direct or indirect beta-sympathomimetic effects
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Modulation of thromboxane (TXA2) and prostacyclin (PGI2) synthesis
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Complement system inhibition
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Antioxidant and free radical scavenging properties
Additional Pharmacological Properties
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Vascular stabilizing (flavonoids)
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Cardioprotective
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Peripheral vascular effects (e.g., skeletal muscle circulation)
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Lipid-lowering effects (increased LDL receptor expression in the liver, inhibition of cholesterol synthesis)
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Anti-inflammatory
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Antioxidant
Clinical Use
May be given in addition to conventional medication.
Long-term use is necessary for sustained benefit.
Indications
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Heart failure stages I and II according to NYHA, possibly also stage III
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Prevention of atherosclerosis
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Cardiac arrhythmias
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Coronary ischemia
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Myocardial weakness after infectious diseases
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Post-myocardial infarction recovery
NYHA Heart Failure Classification
Stage I: No symptoms, normal physical capacity
Stage II: Mild to moderate limitation (e.g., shortness of breath when climbing stairs)
Stage III: Symptoms with mild exertion (e.g., shortness of breath when walking normally), comfortable at rest
Stage IV: Symptoms at rest, bedridden
Contraindications
None known.
Side Effects
Very rare mild gastrointestinal discomfort.
Preparations
Tea, capsules, tablets, tinctures, liquid extracts.
Commercial preparations include:
Crataegutt (drops), Crataegus Verla, Craegium, and others.
